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Brazilian Journal of Nephrology
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Documentos disponibles con este título uniforme (9)
Clasificado(s) por (Año de edición descendente) Refinar búsquedaHemolytic uremic syndrome caused by Shiga toxin-producing Escherichia coli in a renal transplant recipient case report / John Fredy Nieto Ríos ; Mónica Zuluaga Quintero ; Arbey Aristizabal Álzate ; Gustavo Adolfo Zuluaga Valencia
Título : Hemolytic uremic syndrome caused by Shiga toxin-producing Escherichia coli in a renal transplant recipient case report Tipo de documento : documento electrónico Autores : John Fredy Nieto Ríos, ; Mónica Zuluaga Quintero, ; Arbey Aristizabal Álzate, ; Gustavo Adolfo Zuluaga Valencia, Fecha de publicación : 2021 Títulos uniformes : Brazilian Journal of Nephrology Idioma : Inglés (eng) Palabras clave : Thrombotic Microangiopathies Hemolytic-Uremic Syndrome Shiga Toxin Kidney Transplantation ADAMTS13 Protein Alternative Complement Pathway Resumen : Thrombotic microangiopathies are disorders characterized by nonimmune microangiopathic hemolytic anemia, thrombocytopenia, and multi-systemic failure. They are classified as thrombotic thrombocytopenic purpura, atypical hemolytic-uremic syndrome, and typical hemolytic uremic syndrome. The latter is associated with intestinal infections by Shiga toxin-producing bacteria. Typical hemolytic uremic syndrome in adults is an extremely rare condition, characterized by high morbidity and mortality. It has been seldom described in solid organ transplant recipients. Here is presented the case of a kidney transplant recipient who had typical hemolytic uremic syndrome with multisystem commitment, refractory to management and with a fatal outcome. Mención de responsabilidad : John Fredy Nieto-Rios, Monica Zuluaga-Quintero, Julio Cesar Valencia-Maturana, Diana Carolina Bello-Marquez, Arbey Aristizabal-Alzate, Gustavo Adolfo Zuluaga-Valencia, Lina Maria Serna-Higuita, Luis Fernando Arias Referencia : J Bras Nefrol. Oct-Dec 2021;43(4):591-596. DOI (Digital Object Identifier) : 10.1590/2175-8239-jbn-2020-0048 PMID : 33179720 Derechos de uso : CC BY En línea : https://bjnephrology.org/en/article/hemolytic-uremic-syndrome-caused-by-shiga-to [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=5737 Hemolytic uremic syndrome caused by Shiga toxin-producing Escherichia coli in a renal transplant recipient case report [documento electrónico] / John Fredy Nieto Ríos, ; Mónica Zuluaga Quintero, ; Arbey Aristizabal Álzate, ; Gustavo Adolfo Zuluaga Valencia, . - 2021.
Obra : Brazilian Journal of Nephrology
Idioma : Inglés (eng)
Palabras clave : Thrombotic Microangiopathies Hemolytic-Uremic Syndrome Shiga Toxin Kidney Transplantation ADAMTS13 Protein Alternative Complement Pathway Resumen : Thrombotic microangiopathies are disorders characterized by nonimmune microangiopathic hemolytic anemia, thrombocytopenia, and multi-systemic failure. They are classified as thrombotic thrombocytopenic purpura, atypical hemolytic-uremic syndrome, and typical hemolytic uremic syndrome. The latter is associated with intestinal infections by Shiga toxin-producing bacteria. Typical hemolytic uremic syndrome in adults is an extremely rare condition, characterized by high morbidity and mortality. It has been seldom described in solid organ transplant recipients. Here is presented the case of a kidney transplant recipient who had typical hemolytic uremic syndrome with multisystem commitment, refractory to management and with a fatal outcome. Mención de responsabilidad : John Fredy Nieto-Rios, Monica Zuluaga-Quintero, Julio Cesar Valencia-Maturana, Diana Carolina Bello-Marquez, Arbey Aristizabal-Alzate, Gustavo Adolfo Zuluaga-Valencia, Lina Maria Serna-Higuita, Luis Fernando Arias Referencia : J Bras Nefrol. Oct-Dec 2021;43(4):591-596. DOI (Digital Object Identifier) : 10.1590/2175-8239-jbn-2020-0048 PMID : 33179720 Derechos de uso : CC BY En línea : https://bjnephrology.org/en/article/hemolytic-uremic-syndrome-caused-by-shiga-to [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=5737 Reserva
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Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD001676 AC-2020-147 Archivo digital Producción Científica Artículos científicos Disponible Documentos electrónicos
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Título : BK virus nephropathy in a heart transplant recipient Tipo de documento : documento electrónico Autores : John Fredy Nieto Ríos, ; Arbey Aristizabal Álzate, ; Gustavo Adolfo Zuluaga Valencia, Fecha de publicación : 2020 Títulos uniformes : Brazilian Journal of Nephrology Idioma : Inglés (eng) Palabras clave : Polyomavirus BK virus BK Virus Nephropathy Organ Transplantation Heart transplantation Immunosuppression Resumen : BK virus nephropathy in kidney transplantation is widely recognized as an important cause of graft dysfunction and loss. In the case of transplants of organs other than kidney, BK virus nephropathy in native kidneys has been recognized as a cause of chronic kidney disease, which is related with immunosuppression; however, the diagnosis is usually late because the renal dysfunction is attributed to other causes, such as toxicity by anticalcineurinic drugs, interstitial nephritis due to medications, hemodynamic changes, diabetes, hypertension, etc. We report a case of BK virus nephropathy in a patient who underwent heart transplantation due to peripartum cardiomyopathy. The kidney biopsy reported active chronic tubulointerstitial nephritis associated with late stage polyomavirus nephritis and the blood viral load for BK virus was positive (logarithm 4.5). The immunosuppressive treatment was reduced, and after two years of follow-up, the patient had stable renal function with a serum creatinine of 2.5 mg/dL (GFR of 23.4 mL/min/1.73m2). We recommend that the BK virus be considered as a cause of renal dysfunction in heart transplant recipients, with the aim of detecting its replication in time to reduce immunosuppressive therapy before irreversible compromise of renal function may manifest. Mención de responsabilidad : John Fredy Nieto-Ríos, Diego Armando Benavides-Henao, Arbey Aristizabal-Alzate, Carol Morales-Contreras, Diana Carolina Chacón-Jaimes, Gustavo Zuluaga-Valencia, Lina María Serna-Higuita Referencia : J Bras Nefrol. Jul-Sep 2021;43(3):434-439. DOI (Digital Object Identifier) : 10.1590/2175-8239-jbn-2020-0049 PMID : 33527977 Derechos de uso : CC BY En línea : https://bjnephrology.org/en/article/bk-virus-nephropathy-in-a-heart-transplant-r [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=5735 BK virus nephropathy in a heart transplant recipient [documento electrónico] / John Fredy Nieto Ríos, ; Arbey Aristizabal Álzate, ; Gustavo Adolfo Zuluaga Valencia, . - 2020.
Obra : Brazilian Journal of Nephrology
Idioma : Inglés (eng)
Palabras clave : Polyomavirus BK virus BK Virus Nephropathy Organ Transplantation Heart transplantation Immunosuppression Resumen : BK virus nephropathy in kidney transplantation is widely recognized as an important cause of graft dysfunction and loss. In the case of transplants of organs other than kidney, BK virus nephropathy in native kidneys has been recognized as a cause of chronic kidney disease, which is related with immunosuppression; however, the diagnosis is usually late because the renal dysfunction is attributed to other causes, such as toxicity by anticalcineurinic drugs, interstitial nephritis due to medications, hemodynamic changes, diabetes, hypertension, etc. We report a case of BK virus nephropathy in a patient who underwent heart transplantation due to peripartum cardiomyopathy. The kidney biopsy reported active chronic tubulointerstitial nephritis associated with late stage polyomavirus nephritis and the blood viral load for BK virus was positive (logarithm 4.5). The immunosuppressive treatment was reduced, and after two years of follow-up, the patient had stable renal function with a serum creatinine of 2.5 mg/dL (GFR of 23.4 mL/min/1.73m2). We recommend that the BK virus be considered as a cause of renal dysfunction in heart transplant recipients, with the aim of detecting its replication in time to reduce immunosuppressive therapy before irreversible compromise of renal function may manifest. Mención de responsabilidad : John Fredy Nieto-Ríos, Diego Armando Benavides-Henao, Arbey Aristizabal-Alzate, Carol Morales-Contreras, Diana Carolina Chacón-Jaimes, Gustavo Zuluaga-Valencia, Lina María Serna-Higuita Referencia : J Bras Nefrol. Jul-Sep 2021;43(3):434-439. DOI (Digital Object Identifier) : 10.1590/2175-8239-jbn-2020-0049 PMID : 33527977 Derechos de uso : CC BY En línea : https://bjnephrology.org/en/article/bk-virus-nephropathy-in-a-heart-transplant-r [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=5735 Reserva
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Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD001674 AC-2020-145 Archivo digital Producción Científica Artículos científicos Disponible Documentos electrónicos
2020-145Adobe Acrobat PDF
Título : Nephrotic syndrome associated with primary atypical hemolytic uremic syndrome Tipo de documento : documento electrónico Autores : John Fredy Nieto Ríos, Fecha de publicación : 2020 Títulos uniformes : Brazilian Journal of Nephrology Idioma : Inglés (eng) Palabras clave : Atypical Hemolytic Uremic Syndrome Nephrotic Syndrome Acute Renal Injury Hypertension Complement System Proteins Resumen : Primary atypical hemolytic-uremic syndrome is a rare disease characterized by non-immune microangiopathic hemolytic anemia, thrombocytopenia, and renal dysfunction; it is related to alterations in the regulation of the alternative pathway of complement due to genetic mutations. The association with nephrotic syndrome is unusual. We present here a pediatric patient diagnosed with primary atypical hemolytic-uremic syndrome associated with nephrotic syndrome who responded to eculizumab treatment. Mención de responsabilidad : Diana Carolina Bello-Marquez, John Fredy Nieto-Rios, Lina Maria Serna-Higuita, Alfonso Jose Gonzalez-Vergara Referencia : J Bras Nefrol. Jul-Sep 2021;43(3):440-444. DOI (Digital Object Identifier) : 10.1590/2175-8239-jbn-2020-0050 PMID : 32779691 Derechos de uso : CC BY En línea : https://bjnephrology.org/en/article/nephrotic-syndrome-associated-with-primary-a [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=5738 Nephrotic syndrome associated with primary atypical hemolytic uremic syndrome [documento electrónico] / John Fredy Nieto Ríos, . - 2020.
Obra : Brazilian Journal of Nephrology
Idioma : Inglés (eng)
Palabras clave : Atypical Hemolytic Uremic Syndrome Nephrotic Syndrome Acute Renal Injury Hypertension Complement System Proteins Resumen : Primary atypical hemolytic-uremic syndrome is a rare disease characterized by non-immune microangiopathic hemolytic anemia, thrombocytopenia, and renal dysfunction; it is related to alterations in the regulation of the alternative pathway of complement due to genetic mutations. The association with nephrotic syndrome is unusual. We present here a pediatric patient diagnosed with primary atypical hemolytic-uremic syndrome associated with nephrotic syndrome who responded to eculizumab treatment. Mención de responsabilidad : Diana Carolina Bello-Marquez, John Fredy Nieto-Rios, Lina Maria Serna-Higuita, Alfonso Jose Gonzalez-Vergara Referencia : J Bras Nefrol. Jul-Sep 2021;43(3):440-444. DOI (Digital Object Identifier) : 10.1590/2175-8239-jbn-2020-0050 PMID : 32779691 Derechos de uso : CC BY En línea : https://bjnephrology.org/en/article/nephrotic-syndrome-associated-with-primary-a [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=5738 Reserva
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Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD001677 AC-2020-148 Archivo digital Producción Científica Artículos científicos Disponible Documentos electrónicos
2020-148Adobe Acrobat PDF
Título : Successful multiple-exchange peritoneal dialysis in a patient with severe hematological toxicity by methotrexate: case report and literature review Tipo de documento : documento electrónico Autores : Arbey Aristizabal Álzate, ; John Fredy Nieto Ríos, ; Catalina Ocampo Kohn, ; Gustavo Adolfo Zuluaga Valencia, Fecha de publicación : 2019 Títulos uniformes : Brazilian Journal of Nephrology Idioma : Inglés (eng) Palabras clave : Methotrexate pancytopenia renal insufficiency peritoneal dialysis Resumen : Methotrexate is an effective medication to control several diseases; however, it can be very toxic, being myelosuppression one of its main adverse effects, which increases in severity and frequency in patients with renal failure. We present the case of a 68-year-old man with chronic, end-stage renal disease associated with ANCA vasculitis, under treatment with peritoneal dialysis, who received the medication at a low dose, indicated by disease activity, which presented as a complication with severe pancytopenia with mucositis that improved with support measures and multiple-exchange peritoneal dialysis. We reviewed 20 cases published to date of pancytopenia associated with methotrexate in patients on dialysis and found high morbidity and mortality, which is why its use in this type of patient is not recommended. However, when this complication occurs, a therapeutic option could be the use of multiple-exchange peritoneal dialysis in addition to supportive therapy for drug-related toxicity, although it is recognized that studies are required to show the role of multiple-exchange peritoneal dialysis in the removal of this medication. Mención de responsabilidad : Arbey Aristizabal-Alzate, John Fredy Nieto-Rios, Catalina Ocampo-Kohn, Lina Maria Serna-Higuita, Diana Carolina Bello-Marquez, Gustavo Adolfo Zuluaga-Valencia Referencia : J Bras Nefrol. 2018 Sep 21. pii: S0101-28002018005003402. DOI (Digital Object Identifier) : 10.1590/2175-8239-JBN-2018-0095 PMID : 30281061 Derechos de uso : CC BY En línea : http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0101-28002018005003402&l [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4133 Successful multiple-exchange peritoneal dialysis in a patient with severe hematological toxicity by methotrexate: case report and literature review [documento electrónico] / Arbey Aristizabal Álzate, ; John Fredy Nieto Ríos, ; Catalina Ocampo Kohn, ; Gustavo Adolfo Zuluaga Valencia, . - 2019.
Obra : Brazilian Journal of Nephrology
Idioma : Inglés (eng)
Palabras clave : Methotrexate pancytopenia renal insufficiency peritoneal dialysis Resumen : Methotrexate is an effective medication to control several diseases; however, it can be very toxic, being myelosuppression one of its main adverse effects, which increases in severity and frequency in patients with renal failure. We present the case of a 68-year-old man with chronic, end-stage renal disease associated with ANCA vasculitis, under treatment with peritoneal dialysis, who received the medication at a low dose, indicated by disease activity, which presented as a complication with severe pancytopenia with mucositis that improved with support measures and multiple-exchange peritoneal dialysis. We reviewed 20 cases published to date of pancytopenia associated with methotrexate in patients on dialysis and found high morbidity and mortality, which is why its use in this type of patient is not recommended. However, when this complication occurs, a therapeutic option could be the use of multiple-exchange peritoneal dialysis in addition to supportive therapy for drug-related toxicity, although it is recognized that studies are required to show the role of multiple-exchange peritoneal dialysis in the removal of this medication. Mención de responsabilidad : Arbey Aristizabal-Alzate, John Fredy Nieto-Rios, Catalina Ocampo-Kohn, Lina Maria Serna-Higuita, Diana Carolina Bello-Marquez, Gustavo Adolfo Zuluaga-Valencia Referencia : J Bras Nefrol. 2018 Sep 21. pii: S0101-28002018005003402. DOI (Digital Object Identifier) : 10.1590/2175-8239-JBN-2018-0095 PMID : 30281061 Derechos de uso : CC BY En línea : http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0101-28002018005003402&l [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4133 Reserva
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Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD000744 AC-2018-031 Archivo digital Producción Científica Artículos científicos Disponible Documentos electrónicos
2018-031.pdfAdobe Acrobat PDF
Título : Primary hiperoxaluria diagnosed after kidney transplantation: report of 2 cases and literature review Tipo de documento : documento electrónico Autores : John Fredy Nieto Ríos, ; Catalina Ocampo Kohn, ; Gustavo Adolfo Zuluaga Valencia, ; Arbey Aristizabal Álzate, Fecha de publicación : 2017 Títulos uniformes : Brazilian Journal of Nephrology Idioma : Inglés (eng) Palabras clave : Hyperoxaluria primary kidney failure chronic kidney transplantation lithiasis nephrocalcinosis Resumen : Primary hyperoxaluria (PH) is a very rare genetic disorder; it is characterized by total or partial deficiency of the enzymes related to the metabolism of glyoxylate, with an overproduction of calcium oxalate that is deposited in different organs, mainly the kidney, leading to recurrent lithiasis, nephrocalcinosis and end stage renal disease (ESRD). In patients with ESRD that receive kidney transplantation alone, the disease has a relapse of 100%, with graft loss in a high percentage of patients in the first 5 years of transplantation. Three molecular disorders have been described in PH: mutation of the gene alanin glioxalate aminotransferase (AGXT); glyoxalate reductase/hydroxy pyruvate reductase (GRHPR) and 4-OH-2-oxoglutarate aldolase (HOGA1). We present two cases of patients with a history of renal lithiasis who were diagnosed with primary hyperoxaluria in the post-transplant period, manifested by early graft failure, with evidence of calcium oxalate crystals in renal biopsy, hyperoxaluria, hyperoxalemia, and genetic test compatible; they were managed with proper diet, abundant oral liquids, pyridoxine, hydrochlorothiazide and potassium citrate; however, they had slow but progressive deterioration of their grafts function until they reached end-stage chronic renal disease. Mención de responsabilidad : John Fredy Nieto Rios, Monica Zuluaga, Lina Maria Serna Higuita, Adriana Florez, Diana Carolina Bello-Marquez, Arbey Aristizábal, Catalina Ocampo Kohn, Gustavo Adolfo Zuluaga Referencia : J Bras Nefrol. 2017 Oct-Dec;39(4):462-466. DOI (Digital Object Identifier) : 10.5935/0101-2800.20170081 PMID : 29319775 Derechos de uso : CC BY En línea : http://bjn.org.br/about-the-authors/1996/en-US Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4072 Primary hiperoxaluria diagnosed after kidney transplantation: report of 2 cases and literature review [documento electrónico] / John Fredy Nieto Ríos, ; Catalina Ocampo Kohn, ; Gustavo Adolfo Zuluaga Valencia, ; Arbey Aristizabal Álzate, . - 2017.
Obra : Brazilian Journal of Nephrology
Idioma : Inglés (eng)
Palabras clave : Hyperoxaluria primary kidney failure chronic kidney transplantation lithiasis nephrocalcinosis Resumen : Primary hyperoxaluria (PH) is a very rare genetic disorder; it is characterized by total or partial deficiency of the enzymes related to the metabolism of glyoxylate, with an overproduction of calcium oxalate that is deposited in different organs, mainly the kidney, leading to recurrent lithiasis, nephrocalcinosis and end stage renal disease (ESRD). In patients with ESRD that receive kidney transplantation alone, the disease has a relapse of 100%, with graft loss in a high percentage of patients in the first 5 years of transplantation. Three molecular disorders have been described in PH: mutation of the gene alanin glioxalate aminotransferase (AGXT); glyoxalate reductase/hydroxy pyruvate reductase (GRHPR) and 4-OH-2-oxoglutarate aldolase (HOGA1). We present two cases of patients with a history of renal lithiasis who were diagnosed with primary hyperoxaluria in the post-transplant period, manifested by early graft failure, with evidence of calcium oxalate crystals in renal biopsy, hyperoxaluria, hyperoxalemia, and genetic test compatible; they were managed with proper diet, abundant oral liquids, pyridoxine, hydrochlorothiazide and potassium citrate; however, they had slow but progressive deterioration of their grafts function until they reached end-stage chronic renal disease. Mención de responsabilidad : John Fredy Nieto Rios, Monica Zuluaga, Lina Maria Serna Higuita, Adriana Florez, Diana Carolina Bello-Marquez, Arbey Aristizábal, Catalina Ocampo Kohn, Gustavo Adolfo Zuluaga Referencia : J Bras Nefrol. 2017 Oct-Dec;39(4):462-466. DOI (Digital Object Identifier) : 10.5935/0101-2800.20170081 PMID : 29319775 Derechos de uso : CC BY En línea : http://bjn.org.br/about-the-authors/1996/en-US Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4072 Reserva
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Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD000676 AC-2017-065 Archivo digital Producción Científica Artículos científicos Disponible Documentos electrónicos
2017-065.pdfAdobe Acrobat PDF
