Inicio
Resultado de la búsqueda
1 búsqueda de la palabra clave 'crinophagy'
Clasificado(s) por (Año de edición descendente) Refinar búsqueda Genera el flujo rss de la búsqueda
Enlace permanente de la investigación
Título : Autophagy in the endocrine glands Tipo de documento : documento electrónico Autores : Luis Vicente Syro Moreno, Fecha de publicación : 2014 Títulos uniformes : Journal of Molecular Endocrinology Idioma : Inglés (eng) Palabras clave : Autophagy endocrine glands crinophagy endocrine diseases Resumen : Autophagy is an important cellular process involving the degradation of intracellular components. Its regulation is complex and while there are many methods available, there is currently no single effective way of detecting and monitoring autophagy. It has several cellular functions that are conserved throughout the body, as well as a variety of different physiological roles depending on the context of its occurrence in the body. Autophagy is also involved in the pathology of a wide range of diseases. Within the endocrine system, autophagy has both its traditional conserved functions and specific functions. In the endocrine glands, autophagy plays a critical role in controlling intracellular hormone levels. In peptide-secreting cells of glands such as the pituitary gland, crinophagy, a specific form of autophagy, targets the secretory granules to control the levels of stored hormone. In steroid-secreting cells of glands such as the testes and adrenal gland, autophagy targets the steroid-producing organelles. The dysregulation of autophagy in the endocrine glands leads to several different endocrine diseases such as diabetes and infertility. This review aims to clarify the known roles of autophagy in the physiology of the endocrine system, as well as in various endocrine diseases. Mención de responsabilidad : Andrea Weckman, Antonio Di Ieva, Fabio Rotondo, Luis V Syro, Leon D Ortiz, Kalman Kovacs and Michael D Cusimano Referencia : Jour of Mol Endocrin. 2014;52(2):151-63. DOI (Digital Object Identifier) : 10.1530/JME-13-0241 En línea : https://jme.bioscientifica.com/view/journals/jme/52/2/R151.xml Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=3794 Autophagy in the endocrine glands [documento electrónico] / Luis Vicente Syro Moreno, . - 2014.
Obra : Journal of Molecular Endocrinology
Idioma : Inglés (eng)
Palabras clave : Autophagy endocrine glands crinophagy endocrine diseases Resumen : Autophagy is an important cellular process involving the degradation of intracellular components. Its regulation is complex and while there are many methods available, there is currently no single effective way of detecting and monitoring autophagy. It has several cellular functions that are conserved throughout the body, as well as a variety of different physiological roles depending on the context of its occurrence in the body. Autophagy is also involved in the pathology of a wide range of diseases. Within the endocrine system, autophagy has both its traditional conserved functions and specific functions. In the endocrine glands, autophagy plays a critical role in controlling intracellular hormone levels. In peptide-secreting cells of glands such as the pituitary gland, crinophagy, a specific form of autophagy, targets the secretory granules to control the levels of stored hormone. In steroid-secreting cells of glands such as the testes and adrenal gland, autophagy targets the steroid-producing organelles. The dysregulation of autophagy in the endocrine glands leads to several different endocrine diseases such as diabetes and infertility. This review aims to clarify the known roles of autophagy in the physiology of the endocrine system, as well as in various endocrine diseases. Mención de responsabilidad : Andrea Weckman, Antonio Di Ieva, Fabio Rotondo, Luis V Syro, Leon D Ortiz, Kalman Kovacs and Michael D Cusimano Referencia : Jour of Mol Endocrin. 2014;52(2):151-63. DOI (Digital Object Identifier) : 10.1530/JME-13-0241 En línea : https://jme.bioscientifica.com/view/journals/jme/52/2/R151.xml Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=3794 Reserva
Reservar este documentoEjemplares(1)
Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD000369 AC-2014-035 Archivo digital Producción Científica Artículos científicos Disponible