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Posaconazole therapy for severe abdominal candidiasis: A case report / Ana Lucía Correa Ángel  

Público ISBD Título : Posaconazole therapy for severe abdominal candidiasis: A case report Otros títulos : Tratamiento de un caso de candidiasis abdominal severa con posaconazol Tipo de documento : documento electrónico Autores : Ana Lucía Correa Ángel, Fecha de publicación : 2004 Títulos uniformes : Revista Iberoamericana de Micología Idioma : Inglés (eng) Palabras clave : Candidiasis  Candida albicans  Candida tropicalis  Human  SCH56592  Posaconzole  Fluconazole Resumen : We report the successful treatment of a fluconazole-resistant intra-abdominal Candida infection (Candida albicans and Candida tropicalis) with posaconazole (SCH56592) in a 68-year-old woman with a recent history of intra-abdominal surgery. Mención de responsabilidad : Angela M Tobón, Ana L Correa, Myrtha Arango, Catalina de Bedout, Angela Restrepo Referencia : Rev Iberoam Micol. 2004 Jun;21(2):79-81. PMID : 15538832 En línea : http://www.reviberoammicol.com/2004-21/079081.pdf Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4405 Posaconazole therapy for severe abdominal candidiasis: A case report = Tratamiento de un caso de candidiasis abdominal severa con posaconazol [documento electrónico] / Ana Lucía Correa Ángel,  . - 2004. Obra : Revista Iberoamericana de Micología Idioma : Inglés (eng) Palabras clave : Candidiasis  Candida albicans  Candida tropicalis  Human  SCH56592  Posaconzole  Fluconazole Resumen : We report the successful treatment of a fluconazole-resistant intra-abdominal Candida infection (Candida albicans and Candida tropicalis) with posaconazole (SCH56592) in a 68-year-old woman with a recent history of intra-abdominal surgery. Mención de responsabilidad : Angela M Tobón, Ana L Correa, Myrtha Arango, Catalina de Bedout, Angela Restrepo Referencia : Rev Iberoam Micol. 2004 Jun;21(2):79-81. PMID : 15538832 En línea : http://www.reviberoammicol.com/2004-21/079081.pdf Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4405

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DD000905	AC-2004-008	Archivo digital	Producción Científica	Artículos científicos	Disponible

Prolactin-producing pituitary adenoma associated with prolactin cell hyperplasia / Luis Vicente Syro Moreno  

Público ISBD Título : Prolactin-producing pituitary adenoma associated with prolactin cell hyperplasia Tipo de documento : documento electrónico Autores : Luis Vicente Syro Moreno, Fecha de publicación : 2002 Títulos uniformes : Endocrine Pathology Idioma : Inglés (eng) Palabras clave : Prolactin adenoma  pituitary hyperplasia  human  electron microscopy  immunohistochemistry Resumen : A 24-yr-old woman with amenorrhea, galactorrhea, hyperprolactinemia, and sellar mass underwent transsphenoidal surgery. Histologic, immunohistochemical, and electron microscopic investigation revealed a well-differentiated, sparsely granulated prolactin (PRL) cell adenoma of the pituitary showing conclusive PRL immunoreactivity. In the nontumorous adenohypophysis PRL cell hyperplasia was noted. Marked differences were evident between the neoplastic and hyperplastic areas. The tumor consisted of sparsely granulated PRL cells immunoreactive only for PRL. As demonstrated by immunoelectron microscopy, the hyperplastic are a comprised monohormonal sparsely granulated PRL cells as well as bihormonal mammosomatotrophs immunoreactive for both PRL and growth hormone. The MIB-1 index was higher whereas microvessel density was lower in the adenoma as compared with the hyperplastic area. In addition, the nontumorous area showed lymphocytic infiltration whereas inflammatory reaction was not seen in the adenoma. This case represents a rare association of a PRL cell adenoma and PRL cell hyperplasia. The fact that these two lesions were contiguous in the surgically removed material raises the possibility that hyperplasia can precede and transform into adenoma. Mención de responsabilidad : Sergio Vidal DVM, Eva Horvath PhD, Luis V. Syro MD, Humberto Uribe MD, Sandy Cohen RT & Kalman Kovacs MD, PhD Referencia : Endocr Pathol. Summer 2002;13(2):157-65. DOI (Digital Object Identifier) : 10.1385/EP:13:2:157 PMID : 12165665 En línea : https://link.springer.com/article/10.1385/EP:13:2:157 Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4393 Prolactin-producing pituitary adenoma associated with prolactin cell hyperplasia [documento electrónico] / Luis Vicente Syro Moreno,  . - 2002. Obra : Endocrine Pathology Idioma : Inglés (eng) Palabras clave : Prolactin adenoma  pituitary hyperplasia  human  electron microscopy  immunohistochemistry Resumen : A 24-yr-old woman with amenorrhea, galactorrhea, hyperprolactinemia, and sellar mass underwent transsphenoidal surgery. Histologic, immunohistochemical, and electron microscopic investigation revealed a well-differentiated, sparsely granulated prolactin (PRL) cell adenoma of the pituitary showing conclusive PRL immunoreactivity. In the nontumorous adenohypophysis PRL cell hyperplasia was noted. Marked differences were evident between the neoplastic and hyperplastic areas. The tumor consisted of sparsely granulated PRL cells immunoreactive only for PRL. As demonstrated by immunoelectron microscopy, the hyperplastic are a comprised monohormonal sparsely granulated PRL cells as well as bihormonal mammosomatotrophs immunoreactive for both PRL and growth hormone. The MIB-1 index was higher whereas microvessel density was lower in the adenoma as compared with the hyperplastic area. In addition, the nontumorous area showed lymphocytic infiltration whereas inflammatory reaction was not seen in the adenoma. This case represents a rare association of a PRL cell adenoma and PRL cell hyperplasia. The fact that these two lesions were contiguous in the surgically removed material raises the possibility that hyperplasia can precede and transform into adenoma. Mención de responsabilidad : Sergio Vidal DVM, Eva Horvath PhD, Luis V. Syro MD, Humberto Uribe MD, Sandy Cohen RT & Kalman Kovacs MD, PhD Referencia : Endocr Pathol. Summer 2002;13(2):157-65. DOI (Digital Object Identifier) : 10.1385/EP:13:2:157 PMID : 12165665 En línea : https://link.springer.com/article/10.1385/EP:13:2:157 Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4393

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Ultrastructural and immunoelectron microscopic study of three unusual plurihormonal pituitary adenomas / Luis Vicente Syro Moreno  

Público ISBD Título : Ultrastructural and immunoelectron microscopic study of three unusual plurihormonal pituitary adenomas Tipo de documento : documento electrónico Autores : Luis Vicente Syro Moreno, Fecha de publicación : 1999 Títulos uniformes : Ultrastructural Pathology Idioma : Inglés (eng) Palabras clave : Human  Immunoelectron Microscopy  Pituitary Adenoma  Plurihormonality  Ultrastructure Resumen : Momomorphous pituitary adenomas expressing several hormones by immunocytochemistry are common, whereas adenomas displaying multiple immunoreactivities and consisting of more than one morphologic cell types are rare. Three such unusual pituitary adenomas, surgically removed from two patients with acromegaly and one patient with hyperprolactinemia, were investigated by histology, immunocytochemistry, transmission electron microscopy, as well as immunoelectron microscopy using double immunogold labeling. Immunocytochemistry revealed variable degrees of immunoreactivities for growth hormone (GH), prolactin (PRL), thyroid-stimulating hormone (beta-TSH), and alpha-subunit of glycoprotein hormones in all three tumors. The three adenomas consisted of phenotypically diverse cell populations as documented by transmission electron microscopy. In addition to monohormonal GH cells, immunoelectron microscopy demonstrated numerous cells colocalizing GH and PRL or GH and beta-TSH, and rarely PRL and beta-TSH in tumors of acromegalics. The adenoma causing hyperprolactinemia consisted chiefly of mammosomatotrophs colocalizing PRL and GH, whereas beta-TSH labeling was scant. The three tumors in the study were selected from a cluster of five plurimorphous plurihormonal adenomas received from the same locale where they accounted for an unprecedented 21% of adenomas producing GH and/or PRL. The enhanced susceptibility to develop plurimorphous adenomas of the acidophil cell line may have a genetic basis in the stable population the patients came from. Mención de responsabilidad : J F Molina, L R Espinoza Referencia : Ultrastruct Pathol. May-Jun 1999;23(3):141-8. DOI (Digital Object Identifier) : 10.1080/019131299281635 PMID : 10445280 En línea : https://www.tandfonline.com/doi/abs/10.1080/019131299281635 Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4385 Ultrastructural and immunoelectron microscopic study of three unusual plurihormonal pituitary adenomas [documento electrónico] / Luis Vicente Syro Moreno,  . - 1999. Obra : Ultrastructural Pathology Idioma : Inglés (eng) Palabras clave : Human  Immunoelectron Microscopy  Pituitary Adenoma  Plurihormonality  Ultrastructure Resumen : Momomorphous pituitary adenomas expressing several hormones by immunocytochemistry are common, whereas adenomas displaying multiple immunoreactivities and consisting of more than one morphologic cell types are rare. Three such unusual pituitary adenomas, surgically removed from two patients with acromegaly and one patient with hyperprolactinemia, were investigated by histology, immunocytochemistry, transmission electron microscopy, as well as immunoelectron microscopy using double immunogold labeling. Immunocytochemistry revealed variable degrees of immunoreactivities for growth hormone (GH), prolactin (PRL), thyroid-stimulating hormone (beta-TSH), and alpha-subunit of glycoprotein hormones in all three tumors. The three adenomas consisted of phenotypically diverse cell populations as documented by transmission electron microscopy. In addition to monohormonal GH cells, immunoelectron microscopy demonstrated numerous cells colocalizing GH and PRL or GH and beta-TSH, and rarely PRL and beta-TSH in tumors of acromegalics. The adenoma causing hyperprolactinemia consisted chiefly of mammosomatotrophs colocalizing PRL and GH, whereas beta-TSH labeling was scant. The three tumors in the study were selected from a cluster of five plurimorphous plurihormonal adenomas received from the same locale where they accounted for an unprecedented 21% of adenomas producing GH and/or PRL. The enhanced susceptibility to develop plurimorphous adenomas of the acidophil cell line may have a genetic basis in the stable population the patients came from. Mención de responsabilidad : J F Molina, L R Espinoza Referencia : Ultrastruct Pathol. May-Jun 1999;23(3):141-8. DOI (Digital Object Identifier) : 10.1080/019131299281635 PMID : 10445280 En línea : https://www.tandfonline.com/doi/abs/10.1080/019131299281635 Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4385

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