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Endoglin (CD105) : a review of its role in angiogenesis and tumor diagnosis, progression and therapy / Luis Vicente Syro Moreno
Título : Endoglin (CD105) : a review of its role in angiogenesis and tumor diagnosis, progression and therapy Tipo de documento : documento electrónico Autores : Luis Vicente Syro Moreno, Fecha de publicación : 2011 Títulos uniformes : Anticancer Research Idioma : Inglés (eng) Palabras clave : Angiogenesis cancer CD105 endoglin tumor review Resumen : Endoglin (CD105) is an accessory receptor for transforming growth factor beta (TGF-β) and its expression is up-regulated in actively proliferating endothelial cells. Endoglin has been suggested as an appropriate marker for tumor-related angiogenesis and neovascularization. Several studies demonstrate the potential of endoglin in tumor diagnosis, prognosis, and therapy. This review details the structure and function of endoglin, and investigates the role of endoglin in angiogenesis and tumor diagnosis, prognosis, and therapy. Mención de responsabilidad : Farshad Nassiri, Michael D Cusimano, Bernd W Scheithauer, Fabio Rotondo, Alessandra Fazio, George M Yousef, Luis V Syro, Kalman Kovacs, Ricardo V Lloyd Referencia : Anticancer Res. 2011 Jun;31(6):2283-90. PMID : 21737653 En línea : http://ar.iiarjournals.org/content/31/6/2283.full Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=3555 Endoglin (CD105) : a review of its role in angiogenesis and tumor diagnosis, progression and therapy [documento electrónico] / Luis Vicente Syro Moreno, . - 2011.
Obra : Anticancer Research
Idioma : Inglés (eng)
Palabras clave : Angiogenesis cancer CD105 endoglin tumor review Resumen : Endoglin (CD105) is an accessory receptor for transforming growth factor beta (TGF-β) and its expression is up-regulated in actively proliferating endothelial cells. Endoglin has been suggested as an appropriate marker for tumor-related angiogenesis and neovascularization. Several studies demonstrate the potential of endoglin in tumor diagnosis, prognosis, and therapy. This review details the structure and function of endoglin, and investigates the role of endoglin in angiogenesis and tumor diagnosis, prognosis, and therapy. Mención de responsabilidad : Farshad Nassiri, Michael D Cusimano, Bernd W Scheithauer, Fabio Rotondo, Alessandra Fazio, George M Yousef, Luis V Syro, Kalman Kovacs, Ricardo V Lloyd Referencia : Anticancer Res. 2011 Jun;31(6):2283-90. PMID : 21737653 En línea : http://ar.iiarjournals.org/content/31/6/2283.full Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=3555 Reserva
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Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD000124 AC-2011-009 Archivo digital Producción Científica Artículos científicos Disponible Endoglin and CD-34 immunoreactivity in the assessment of microvessel density in normal pituitary and adenoma subtypes / Luis Vicente Syro Moreno
Título : Endoglin and CD-34 immunoreactivity in the assessment of microvessel density in normal pituitary and adenoma subtypes Tipo de documento : documento electrónico Autores : Luis Vicente Syro Moreno, Fecha de publicación : 2010 Títulos uniformes : Neoplasma Idioma : Inglés (eng) Palabras clave : immunohistochemistry endoglin CD34 microvascular density angiogenesis pituitary Resumen : Vascularization is a prerequisite of tumor growth, invasion and metastasis. In the present work, microvessel density was assessed by quantitating using two different endothelial cell biomarkers, endoglin (CD-105) and CD-34. Fifty endocrinologically active and 36 clinically nonfunctioning pituitary adenomas, all surgically resected, as well as 10 autopsy-derived normal adenohypophyses were investigated by immunohistochemistry. The results showed that in every pituitary adenoma type endoglin, an assumed biomarker of proliferating endothelial cells, immunostained fewer vessels than CD-34 which revealed immunopositivity in all capillaries. Differences in endoglin versus CD-34 immunoexpression indicate varying degrees of vascularity in pituitary adenoma subtypes. The low levels of endoglin immunoexpression in pituitary tumors exposed to long-acting somatostatin analogs and dopamine agonists are consistent with the view that these agents inhibit angiogenesis. Mención de responsabilidad : F Rotondo, S Sharma, B W Scheithauer, E Horvath, L V Syro, M Cusimano, F Nassiri, G M Yousef, K Kovacs Referencia : Neoplasma. 2010;57(6):590-3. DOI (Digital Object Identifier) : 10.4149/neo_2010_06_590 PMID : 20845998 En línea : http://www.elis.sk/index.php?page=shop.product_details&flypage=flypage.tpl&produ [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4470 Endoglin and CD-34 immunoreactivity in the assessment of microvessel density in normal pituitary and adenoma subtypes [documento electrónico] / Luis Vicente Syro Moreno, . - 2010.
Obra : Neoplasma
Idioma : Inglés (eng)
Palabras clave : immunohistochemistry endoglin CD34 microvascular density angiogenesis pituitary Resumen : Vascularization is a prerequisite of tumor growth, invasion and metastasis. In the present work, microvessel density was assessed by quantitating using two different endothelial cell biomarkers, endoglin (CD-105) and CD-34. Fifty endocrinologically active and 36 clinically nonfunctioning pituitary adenomas, all surgically resected, as well as 10 autopsy-derived normal adenohypophyses were investigated by immunohistochemistry. The results showed that in every pituitary adenoma type endoglin, an assumed biomarker of proliferating endothelial cells, immunostained fewer vessels than CD-34 which revealed immunopositivity in all capillaries. Differences in endoglin versus CD-34 immunoexpression indicate varying degrees of vascularity in pituitary adenoma subtypes. The low levels of endoglin immunoexpression in pituitary tumors exposed to long-acting somatostatin analogs and dopamine agonists are consistent with the view that these agents inhibit angiogenesis. Mención de responsabilidad : F Rotondo, S Sharma, B W Scheithauer, E Horvath, L V Syro, M Cusimano, F Nassiri, G M Yousef, K Kovacs Referencia : Neoplasma. 2010;57(6):590-3. DOI (Digital Object Identifier) : 10.4149/neo_2010_06_590 PMID : 20845998 En línea : http://www.elis.sk/index.php?page=shop.product_details&flypage=flypage.tpl&produ [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4470 Reserva
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Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD000970 AC-2010-060 Archivo digital Producción Científica Artículos científicos Disponible