Inicio
Resultado de la búsqueda
8 búsqueda de la palabra clave 'kidney transplantation'
Clasificado(s) por (Año de edición descendente) Refinar búsqueda Genera el flujo rss de la búsqueda
Enlace permanente de la investigaciónHemolytic uremic syndrome caused by Shiga toxin-producing Escherichia coli in a renal transplant recipient case report / John Fredy Nieto Ríos ; Mónica Zuluaga Quintero ; Arbey Aristizabal Álzate ; Gustavo Adolfo Zuluaga Valencia
Título : Hemolytic uremic syndrome caused by Shiga toxin-producing Escherichia coli in a renal transplant recipient case report Tipo de documento : documento electrónico Autores : John Fredy Nieto Ríos, ; Mónica Zuluaga Quintero, ; Arbey Aristizabal Álzate, ; Gustavo Adolfo Zuluaga Valencia, Fecha de publicación : 2021 Títulos uniformes : Brazilian Journal of Nephrology Idioma : Inglés (eng) Palabras clave : Thrombotic Microangiopathies Hemolytic-Uremic Syndrome Shiga Toxin Kidney Transplantation ADAMTS13 Protein Alternative Complement Pathway Resumen : Thrombotic microangiopathies are disorders characterized by nonimmune microangiopathic hemolytic anemia, thrombocytopenia, and multi-systemic failure. They are classified as thrombotic thrombocytopenic purpura, atypical hemolytic-uremic syndrome, and typical hemolytic uremic syndrome. The latter is associated with intestinal infections by Shiga toxin-producing bacteria. Typical hemolytic uremic syndrome in adults is an extremely rare condition, characterized by high morbidity and mortality. It has been seldom described in solid organ transplant recipients. Here is presented the case of a kidney transplant recipient who had typical hemolytic uremic syndrome with multisystem commitment, refractory to management and with a fatal outcome. Mención de responsabilidad : John Fredy Nieto-Rios, Monica Zuluaga-Quintero, Julio Cesar Valencia-Maturana, Diana Carolina Bello-Marquez, Arbey Aristizabal-Alzate, Gustavo Adolfo Zuluaga-Valencia, Lina Maria Serna-Higuita, Luis Fernando Arias Referencia : J Bras Nefrol. Oct-Dec 2021;43(4):591-596. DOI (Digital Object Identifier) : 10.1590/2175-8239-jbn-2020-0048 PMID : 33179720 Derechos de uso : CC BY En línea : https://bjnephrology.org/en/article/hemolytic-uremic-syndrome-caused-by-shiga-to [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=5737 Hemolytic uremic syndrome caused by Shiga toxin-producing Escherichia coli in a renal transplant recipient case report [documento electrónico] / John Fredy Nieto Ríos, ; Mónica Zuluaga Quintero, ; Arbey Aristizabal Álzate, ; Gustavo Adolfo Zuluaga Valencia, . - 2021.
Obra : Brazilian Journal of Nephrology
Idioma : Inglés (eng)
Palabras clave : Thrombotic Microangiopathies Hemolytic-Uremic Syndrome Shiga Toxin Kidney Transplantation ADAMTS13 Protein Alternative Complement Pathway Resumen : Thrombotic microangiopathies are disorders characterized by nonimmune microangiopathic hemolytic anemia, thrombocytopenia, and multi-systemic failure. They are classified as thrombotic thrombocytopenic purpura, atypical hemolytic-uremic syndrome, and typical hemolytic uremic syndrome. The latter is associated with intestinal infections by Shiga toxin-producing bacteria. Typical hemolytic uremic syndrome in adults is an extremely rare condition, characterized by high morbidity and mortality. It has been seldom described in solid organ transplant recipients. Here is presented the case of a kidney transplant recipient who had typical hemolytic uremic syndrome with multisystem commitment, refractory to management and with a fatal outcome. Mención de responsabilidad : John Fredy Nieto-Rios, Monica Zuluaga-Quintero, Julio Cesar Valencia-Maturana, Diana Carolina Bello-Marquez, Arbey Aristizabal-Alzate, Gustavo Adolfo Zuluaga-Valencia, Lina Maria Serna-Higuita, Luis Fernando Arias Referencia : J Bras Nefrol. Oct-Dec 2021;43(4):591-596. DOI (Digital Object Identifier) : 10.1590/2175-8239-jbn-2020-0048 PMID : 33179720 Derechos de uso : CC BY En línea : https://bjnephrology.org/en/article/hemolytic-uremic-syndrome-caused-by-shiga-to [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=5737 Reserva
Reservar este documentoEjemplares(1)
Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD001676 AC-2020-147 Archivo digital Producción Científica Artículos científicos Disponible Documentos electrónicos
2020-147Adobe Acrobat PDF
Título : Treatment of post-biopsy arteriovenous fistula of a renal graft by selective embolization Tipo de documento : documento electrónico Autores : José Miguel Hidalgo Oviedo, ; Sergio Álvarez Vallejo, ; Arbey Aristizabal Álzate, ; Gustavo Adolfo Zuluaga Valencia, ; John Fredy Nieto Ríos, Fecha de publicación : 2021 Títulos uniformes : Indian Journal of Nephrology Idioma : Inglés (eng) Palabras clave : Arteriovenous fistula kidney transplantation graft biopsy graft rejection selective embolization Resumen : The development of an arteriovenous fistula (AVF) after renal graft biopsy is a rare complication, it is associated in most cases with spontaneous resolution. However, interventional therapies are required in some cases, to prevent graft loss. Selective embolization has been described as an alternative treatment. In the present study, we describes our experience on AVF after biopsy in kidney transplant patients, which was managed with selective embolization. From 2005 to 2015, a total of 452 kidney transplant biopsies were performed, 12 had an AVF requiring embolization. In 92% of cases, this was successful. Beforehand, mean serum creatinine levels were 2.45 mg/dL, after the procedure, that increased to 3.05, however, 3 months later, mean creatinine levels dropped to 1.85 mg/dL. Graft survival after 2 follow-up years was 72%. Our experience demonstrates that selective embolization of the AVF after kidney transplant biopsy is a safe procedure, and that transplant function can be maintained in patients with this complication. Mención de responsabilidad : Lina M Serna-Higuita, Monica Zuluaga-Quintero, Jose M Hidalgo-Oviedo, Sergio Alvarez Vallejo, Arbey Aristizabal-Alzate, Gustavo A Zuluaga-Valencia, John F Nieto-Ríos Referencia : Indian J Nephrol. Mar-Apr 2021;31(2):201-204. DOI (Digital Object Identifier) : 10.4103/ijn.IJN_351_19 PMID : 34267449 En línea : https://www.indianjnephrol.org/article.asp?issn=0971-4065;year=2021;volume=31;is [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=5777 Treatment of post-biopsy arteriovenous fistula of a renal graft by selective embolization [documento electrónico] / José Miguel Hidalgo Oviedo, ; Sergio Álvarez Vallejo, ; Arbey Aristizabal Álzate, ; Gustavo Adolfo Zuluaga Valencia, ; John Fredy Nieto Ríos, . - 2021.
Obra : Indian Journal of Nephrology
Idioma : Inglés (eng)
Palabras clave : Arteriovenous fistula kidney transplantation graft biopsy graft rejection selective embolization Resumen : The development of an arteriovenous fistula (AVF) after renal graft biopsy is a rare complication, it is associated in most cases with spontaneous resolution. However, interventional therapies are required in some cases, to prevent graft loss. Selective embolization has been described as an alternative treatment. In the present study, we describes our experience on AVF after biopsy in kidney transplant patients, which was managed with selective embolization. From 2005 to 2015, a total of 452 kidney transplant biopsies were performed, 12 had an AVF requiring embolization. In 92% of cases, this was successful. Beforehand, mean serum creatinine levels were 2.45 mg/dL, after the procedure, that increased to 3.05, however, 3 months later, mean creatinine levels dropped to 1.85 mg/dL. Graft survival after 2 follow-up years was 72%. Our experience demonstrates that selective embolization of the AVF after kidney transplant biopsy is a safe procedure, and that transplant function can be maintained in patients with this complication. Mención de responsabilidad : Lina M Serna-Higuita, Monica Zuluaga-Quintero, Jose M Hidalgo-Oviedo, Sergio Alvarez Vallejo, Arbey Aristizabal-Alzate, Gustavo A Zuluaga-Valencia, John F Nieto-Ríos Referencia : Indian J Nephrol. Mar-Apr 2021;31(2):201-204. DOI (Digital Object Identifier) : 10.4103/ijn.IJN_351_19 PMID : 34267449 En línea : https://www.indianjnephrol.org/article.asp?issn=0971-4065;year=2021;volume=31;is [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=5777 Reserva
Reservar este documentoEjemplares(1)
Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD001704 AC-2021-027 Archivo digital Producción Científica Artículos científicos Disponible
Título : Time of Cold Ischemia and Delayed Graft Function in a Cohort of Renal Transplant Patients in a Reference Center Tipo de documento : documento electrónico Autores : John Fredy Nieto Ríos, ; Catalina Ocampo Kohn, ; Carolina Lucía Ochoa García, ; Arbey Aristizabal Álzate, ; Gustavo Adolfo Zuluaga Valencia, Fecha de publicación : 2019 Títulos uniformes : Indian Journal of Nephrology Idioma : Inglés (eng) Palabras clave : Acute rejection cold ischemia time delayed graft function graft loss immunosuppression kidney transplantation Resumen : There are many factors involved in the delayed graft function of a renal graft, with prolonged cold ischemia time being one of the most relevant. The aim of this study is to evaluate the relationship between the time of cold ischemia and the delayed graft function, and acute rejection and graft loss at 1 year of follow-up. A retrospective cohort of 347 renal transplant patients were evaluated during the years 2009-2013. The incidence of delayed graft function was 18.4% (n = 65). The cold ischemia time was an independent risk factor for delayed graft function (odds ratio [OR] 1.10, 95% confidence interval [CI] 1.04-1.16). By grouping the time of cold ischemia by intervals, the risk of delayed graft function was greater in the 12-18 hours group (OR 2.06, 95% CI 1.02-4.15) and in the >18 hours group (OR 3.38, 95% CI 1.57-7.27). The risk of acute rejection did not increase with longer cold ischemia (p = 0.69), and cold ischemia time was not a risk factor for renal graft loss at 1-year follow-up (hazard ratio 0.97, 95% CI 0.88-1.06). In conclusion the time of cold ischemia (>12 hours) in renal transplant recipients of optimal deceased donors increases the risk of delayed graft function; however, this does not negatively impact the results in acute rejection or graft loss in the first year of the transplant. Mención de responsabilidad : J F Nieto-Ríos, C L Ochoa-García, A Serna-Campuzano, B Benavides-Hermosa, L L Calderón-Puentes, A Aristizabal-Alzate, C Ocampo-Kohn, G Zuluaga-Valencia, L M Serna-Higuita Referencia : Indian J Nephrol. 2019 Jan-Feb;29(1):8-14. DOI (Digital Object Identifier) : 10.4103/ijn.IJN_162_18 PMID : 30814787 Derechos de uso : CC BY-NC-SA En línea : http://www.indianjnephrol.org/article.asp?issn=0971-4065;year=2019;volume=29;iss [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4259 Time of Cold Ischemia and Delayed Graft Function in a Cohort of Renal Transplant Patients in a Reference Center [documento electrónico] / John Fredy Nieto Ríos, ; Catalina Ocampo Kohn, ; Carolina Lucía Ochoa García, ; Arbey Aristizabal Álzate, ; Gustavo Adolfo Zuluaga Valencia, . - 2019.
Obra : Indian Journal of Nephrology
Idioma : Inglés (eng)
Palabras clave : Acute rejection cold ischemia time delayed graft function graft loss immunosuppression kidney transplantation Resumen : There are many factors involved in the delayed graft function of a renal graft, with prolonged cold ischemia time being one of the most relevant. The aim of this study is to evaluate the relationship between the time of cold ischemia and the delayed graft function, and acute rejection and graft loss at 1 year of follow-up. A retrospective cohort of 347 renal transplant patients were evaluated during the years 2009-2013. The incidence of delayed graft function was 18.4% (n = 65). The cold ischemia time was an independent risk factor for delayed graft function (odds ratio [OR] 1.10, 95% confidence interval [CI] 1.04-1.16). By grouping the time of cold ischemia by intervals, the risk of delayed graft function was greater in the 12-18 hours group (OR 2.06, 95% CI 1.02-4.15) and in the >18 hours group (OR 3.38, 95% CI 1.57-7.27). The risk of acute rejection did not increase with longer cold ischemia (p = 0.69), and cold ischemia time was not a risk factor for renal graft loss at 1-year follow-up (hazard ratio 0.97, 95% CI 0.88-1.06). In conclusion the time of cold ischemia (>12 hours) in renal transplant recipients of optimal deceased donors increases the risk of delayed graft function; however, this does not negatively impact the results in acute rejection or graft loss in the first year of the transplant. Mención de responsabilidad : J F Nieto-Ríos, C L Ochoa-García, A Serna-Campuzano, B Benavides-Hermosa, L L Calderón-Puentes, A Aristizabal-Alzate, C Ocampo-Kohn, G Zuluaga-Valencia, L M Serna-Higuita Referencia : Indian J Nephrol. 2019 Jan-Feb;29(1):8-14. DOI (Digital Object Identifier) : 10.4103/ijn.IJN_162_18 PMID : 30814787 Derechos de uso : CC BY-NC-SA En línea : http://www.indianjnephrol.org/article.asp?issn=0971-4065;year=2019;volume=29;iss [...] Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4259 Reserva
Reservar este documentoEjemplares(1)
Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD001238 AC-2019-027 Archivo digital Producción Científica Artículos científicos Disponible Documentos electrónicos
2019-027.pdfAdobe Acrobat PDF
Título : Malakoplakia after kidney transplantation: Case report and literature review Tipo de documento : documento electrónico Autores : John Fredy Nieto Ríos, ; Isabel Cristina Ramírez Sánchez, ; Lina Maria Serna Higuita, ; Alejandro Vélez Hoyos, ; Federico Gaviria Gil, Fecha de publicación : 2017 Títulos uniformes : Transplant Infectious Disease Idioma : Inglés (eng) Palabras clave : Escherichia coli immunosuppression kidney transplantation malakoplakia urinary tract infection Resumen : Malakoplakia is a granulomatous disease associated with an infectious etiology, usually involving the urinary tract. It reveals itself as a recurrent urinary tract infection (r‐UTI), and in some cases, it is associated with impairment of renal function. Immunosuppression is one of its main associated factors, and it has been increasingly described in patients with solid organ transplantation (SOT), mainly kidney transplantation. Macroscopically, it can form masses and sometimes it may be confused with neoplasia, which is why histological findings are fundamental for the diagnosis. Here, we present a case of bladder malakoplakia, manifested by r‐UTI from Escherichia coli in a patient with renal transplantation, refractory to long‐term antibiotic treatment and reduction in immunosuppression, which resolved after surgical management. We also summarize the clinical characteristics of malakoplakia and compare them with previous reports in the literature on SOT. Mención de responsabilidad : John Fredy Nieto-Ríos, Isabel Ramírez, Mónica Zuluaga-Quintero, Lina María Serna-Higuita, Federico Gaviria-Gil, Alejandro Velez-Hoyos Referencia : Transpl Infect Dis. 2017 Oct;19(5). DOI (Digital Object Identifier) : 10.1111/tid.12731 PMID : 28561517 En línea : https://onlinelibrary.wiley.com/doi/abs/10.1111/tid.12731 Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4065 Malakoplakia after kidney transplantation: Case report and literature review [documento electrónico] / John Fredy Nieto Ríos, ; Isabel Cristina Ramírez Sánchez, ; Lina Maria Serna Higuita, ; Alejandro Vélez Hoyos, ; Federico Gaviria Gil, . - 2017.
Obra : Transplant Infectious Disease
Idioma : Inglés (eng)
Palabras clave : Escherichia coli immunosuppression kidney transplantation malakoplakia urinary tract infection Resumen : Malakoplakia is a granulomatous disease associated with an infectious etiology, usually involving the urinary tract. It reveals itself as a recurrent urinary tract infection (r‐UTI), and in some cases, it is associated with impairment of renal function. Immunosuppression is one of its main associated factors, and it has been increasingly described in patients with solid organ transplantation (SOT), mainly kidney transplantation. Macroscopically, it can form masses and sometimes it may be confused with neoplasia, which is why histological findings are fundamental for the diagnosis. Here, we present a case of bladder malakoplakia, manifested by r‐UTI from Escherichia coli in a patient with renal transplantation, refractory to long‐term antibiotic treatment and reduction in immunosuppression, which resolved after surgical management. We also summarize the clinical characteristics of malakoplakia and compare them with previous reports in the literature on SOT. Mención de responsabilidad : John Fredy Nieto-Ríos, Isabel Ramírez, Mónica Zuluaga-Quintero, Lina María Serna-Higuita, Federico Gaviria-Gil, Alejandro Velez-Hoyos Referencia : Transpl Infect Dis. 2017 Oct;19(5). DOI (Digital Object Identifier) : 10.1111/tid.12731 PMID : 28561517 En línea : https://onlinelibrary.wiley.com/doi/abs/10.1111/tid.12731 Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4065 Reserva
Reservar este documentoEjemplares(1)
Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD000667 AC-2017-056 Archivo digital Producción Científica Artículos científicos Disponible
Título : Primary hiperoxaluria diagnosed after kidney transplantation: report of 2 cases and literature review Tipo de documento : documento electrónico Autores : John Fredy Nieto Ríos, ; Catalina Ocampo Kohn, ; Gustavo Adolfo Zuluaga Valencia, ; Arbey Aristizabal Álzate, Fecha de publicación : 2017 Títulos uniformes : Brazilian Journal of Nephrology Idioma : Inglés (eng) Palabras clave : Hyperoxaluria primary kidney failure chronic kidney transplantation lithiasis nephrocalcinosis Resumen : Primary hyperoxaluria (PH) is a very rare genetic disorder; it is characterized by total or partial deficiency of the enzymes related to the metabolism of glyoxylate, with an overproduction of calcium oxalate that is deposited in different organs, mainly the kidney, leading to recurrent lithiasis, nephrocalcinosis and end stage renal disease (ESRD). In patients with ESRD that receive kidney transplantation alone, the disease has a relapse of 100%, with graft loss in a high percentage of patients in the first 5 years of transplantation. Three molecular disorders have been described in PH: mutation of the gene alanin glioxalate aminotransferase (AGXT); glyoxalate reductase/hydroxy pyruvate reductase (GRHPR) and 4-OH-2-oxoglutarate aldolase (HOGA1). We present two cases of patients with a history of renal lithiasis who were diagnosed with primary hyperoxaluria in the post-transplant period, manifested by early graft failure, with evidence of calcium oxalate crystals in renal biopsy, hyperoxaluria, hyperoxalemia, and genetic test compatible; they were managed with proper diet, abundant oral liquids, pyridoxine, hydrochlorothiazide and potassium citrate; however, they had slow but progressive deterioration of their grafts function until they reached end-stage chronic renal disease. Mención de responsabilidad : John Fredy Nieto Rios, Monica Zuluaga, Lina Maria Serna Higuita, Adriana Florez, Diana Carolina Bello-Marquez, Arbey Aristizábal, Catalina Ocampo Kohn, Gustavo Adolfo Zuluaga Referencia : J Bras Nefrol. 2017 Oct-Dec;39(4):462-466. DOI (Digital Object Identifier) : 10.5935/0101-2800.20170081 PMID : 29319775 Derechos de uso : CC BY En línea : http://bjn.org.br/about-the-authors/1996/en-US Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4072 Primary hiperoxaluria diagnosed after kidney transplantation: report of 2 cases and literature review [documento electrónico] / John Fredy Nieto Ríos, ; Catalina Ocampo Kohn, ; Gustavo Adolfo Zuluaga Valencia, ; Arbey Aristizabal Álzate, . - 2017.
Obra : Brazilian Journal of Nephrology
Idioma : Inglés (eng)
Palabras clave : Hyperoxaluria primary kidney failure chronic kidney transplantation lithiasis nephrocalcinosis Resumen : Primary hyperoxaluria (PH) is a very rare genetic disorder; it is characterized by total or partial deficiency of the enzymes related to the metabolism of glyoxylate, with an overproduction of calcium oxalate that is deposited in different organs, mainly the kidney, leading to recurrent lithiasis, nephrocalcinosis and end stage renal disease (ESRD). In patients with ESRD that receive kidney transplantation alone, the disease has a relapse of 100%, with graft loss in a high percentage of patients in the first 5 years of transplantation. Three molecular disorders have been described in PH: mutation of the gene alanin glioxalate aminotransferase (AGXT); glyoxalate reductase/hydroxy pyruvate reductase (GRHPR) and 4-OH-2-oxoglutarate aldolase (HOGA1). We present two cases of patients with a history of renal lithiasis who were diagnosed with primary hyperoxaluria in the post-transplant period, manifested by early graft failure, with evidence of calcium oxalate crystals in renal biopsy, hyperoxaluria, hyperoxalemia, and genetic test compatible; they were managed with proper diet, abundant oral liquids, pyridoxine, hydrochlorothiazide and potassium citrate; however, they had slow but progressive deterioration of their grafts function until they reached end-stage chronic renal disease. Mención de responsabilidad : John Fredy Nieto Rios, Monica Zuluaga, Lina Maria Serna Higuita, Adriana Florez, Diana Carolina Bello-Marquez, Arbey Aristizábal, Catalina Ocampo Kohn, Gustavo Adolfo Zuluaga Referencia : J Bras Nefrol. 2017 Oct-Dec;39(4):462-466. DOI (Digital Object Identifier) : 10.5935/0101-2800.20170081 PMID : 29319775 Derechos de uso : CC BY En línea : http://bjn.org.br/about-the-authors/1996/en-US Enlace permanente : https://hospitalpablotobon.cloudbiteca.com/pmb/opac_css/index.php?lvl=notice_display&id=4072 Reserva
Reservar este documentoEjemplares(1)
Código de barras Número de Ubicación Tipo de medio Ubicación Sección Estado DD000676 AC-2017-065 Archivo digital Producción Científica Artículos científicos Disponible Documentos electrónicos
2017-065.pdfAdobe Acrobat PDF
